A point mutation that confers constitutive activity to CXCR4 reveals that T140 is an inverse agonist and that AMD3100 and ALX40-4C are weak partial agonists
Wen Bo Zhang, Jean Marc Navenot, Bodduluri Haribabu, Hirokazu Tamamuraz, Kenichi Hiramatu, Akane Omagari, Gang Pei, John P. Manfredi, Nobutaka Fujii, James R. Broach, Stephen C. Peiper
Research output: Contribution to journal › Article › peer-review
Dive into the research topics of 'A point mutation that confers constitutive activity to CXCR4 reveals that T140 is an inverse agonist and that AMD3100 and ALX40-4C are weak partial agonists'. Together they form a unique fingerprint.