AhR promotes phosphorylation of ARNT isoform 1 in human T cell malignancies as a switch for optimal AhR activity

Keyphrases

• T Cells 100%
• Aryl Hydrocarbon Receptor Activity 100%
• Aryl Hydrocarbon Receptor 100%
• Phosphorylation 100%
• T-cell Malignancies 100%
• Receptor Targets 11%
• Casein Kinase 2 11%
• Gene Expression Pattern 11%
• Target Gene Regulation 11%
• Immune Function 5%
• Molecular Study 5%
• Nuclear Translocation 5%
• Inflammation 5%
• Transcription Factor 5%
• Aryl Hydrocarbon Receptor Signaling 5%
• Heterodimerization 5%
• Phosphorylation Sites 5%
• Genetic Background 5%
• Immunosuppression 5%
• Immune Cells 5%
• Immune System 5%
• Immune Disorders 5%
• Immune Homeostasis 5%
• T-cell Lymphoma 5%
• Isoform Distribution 5%
• Transcriptome Analysis 5%
• Lymphoma Cells 5%
• Short Isoform 5%
• Global Transcriptome 5%
• Ligand-independent Activation 5%
• Targeted Transcriptomics 5%

Biochemistry, Genetics and Molecular Biology

• Aryl Hydrocarbon Receptor 100%
• Aryl Hydrocarbon Receptor Nuclear Translocator 100%
• T Cell 100%
• Isoform 100%
• Gene Expression 16%
• Gene Control 16%
• Casein Kinase 16%
• Casein Kinase 2 16%
• Transcription Factors 8%
• Mediator 8%
• Homeostasis 8%
• Genetic Background 8%
• Gene Expression Profiling 8%
• Immunocompetent Cell 8%