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Apigenin inhibits pancreatic stellate cell activity in pancreatitis
Amy A. Mrazek
, Laura J. Porro
, Vandanajay Bhatia
,
Miriam Falzon
,
Heidi Spratt
,
Jia Zhou
, Celia Chao
, Mark Hellmich
Surgery
Pharmacology & Toxicology
Research output
:
Contribution to journal
›
Article
›
peer-review
37
Scopus citations
Overview
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Keyphrases
Pancreatitis
100%
Cell Activity
100%
Pancreatic Stellate Cells
100%
Apigenin
100%
Recurrent Acute Pancreatitis
44%
Chronic Pancreatitis
33%
Parathyroid Hormone-related Protein (PTHrP)
22%
Fibronectin
22%
Pancreatic Fibrosis
22%
Apoptosis
11%
Induced Apoptosis
11%
Pancreatic Injury
11%
Pancreas
11%
Inflammation
11%
Mechanism of Action
11%
Gene Expression
11%
In Vitro Study
11%
Inflammatory Mediators
11%
Preclinical Testing
11%
Fibrosis
11%
Extracellular Matrix Protein 1 (ECM1)
11%
Quantitative PCR
11%
Interleukin-6
11%
Response to Injury
11%
Dose Effect
11%
Therapeutic Agents
11%
Patient Care
11%
Immunostaining
11%
MRNA Level
11%
Necrosis
11%
Patients at Risk
11%
Proliferating Cell nuclear Antigen
11%
Transforming Growth Factor-β
11%
Messenger RNA Expression
11%
Once-daily
11%
Oral Gavage
11%
Fibrotic Response
11%
Human Pancreatic Stellate Cells
11%
Profibrotic
11%
Organ Fibrosis
11%
Apigenin Analogues
11%
Collagen 1A1
11%
Medicine and Dentistry
Pancreatitis
100%
Cell Activity
100%
Pancreatic Stellate Cell
100%
Apigenin
100%
Acute Pancreatitis
40%
Chronic Pancreatitis
30%
In Vitro
20%
Programmed Cell Death
20%
Parathyroid Hormone-Related Protein
20%
Fibrosis
20%
Cystic Fibrosis
20%
Fibronectin
20%
Gene Expression
10%
Patient Care
10%
Mediator
10%
Therapeutic Agent
10%
Immunohistochemistry
10%
Necrosis
10%
Proliferating Cell Nuclear Antigen
10%
Interleukin 6
10%
Transforming Growth Factor Beta
10%
Ceruletide
10%
Scleroprotein
10%
Feeding
10%
Pancreatic Injury
10%
Messenger RNA
10%
Reverse Transcription Polymerase Chain Reaction
10%
Oral
10%