Aryl hydrocarbon receptor-dependent stanniocalcin 2 induction by cinnabarinic acid provides cytoprotection against endoplasmic reticulum and oxidative stress

Keyphrases

• Oxidative Stress 100%
• Aryl Hydrocarbon Receptor 100%
• Endoplasmic Reticulum Stress (ER stress) 100%
• Cytoprotection 100%
• Stanniocalcin-1 100%
• Cinnabarinic Acid 100%
• Apoptosis 40%
• Hydrogen Peroxide 20%
• Thapsigargin 20%
• Ethanol-induced Liver Injury 10%
• Knock-in Mouse Model 10%
• Mouse Model 10%
• Novel Mechanism 10%
• Transcription Factor 10%
• Receptor Binding 10%
• Receptor-mediated 10%
• Chromatin Immunoprecipitation (ChIP) 10%
• Xenobiotic Response Element 10%
• Cell Surface 10%
• Liver 10%
• RNA Interference 10%
• Liver Injury 10%
• Liver-specific 10%
• Primary Hepatocytes 10%
• In Vivo Data 10%
• Acid Activation 10%
• Paracrine Signaling 10%
• Microvesicular Steatosis 10%
• Acid Treatment 10%
• Endoplasmic Reticulum 10%
• Immunochemistry 10%
• Hepatotoxicity 10%
• Xenobiotic Metabolism 10%
• Acute Alcohol Intoxication 10%
• Surface Consistent 10%
• Floxed 10%
• Tryptophan Catabolites 10%
• Aryl Hydrocarbon Receptor Signaling Pathway 10%

Pharmacology, Toxicology and Pharmaceutical Science

• Aromatic Hydrocarbon Receptor 100%
• Cytoprotection 100%
• Hypocalcin 100%
• Hydrogen Peroxide 27%
• Cytoprotective Agent 18%
• Thapsigargin 18%
• Mouse Model 9%
• Transcription Factors 9%
• Liver Injury 9%
• Steatosis 9%
• Knockout Mouse 9%
• Chromatin Immunoprecipitation 9%
• Tryptophan 9%
• Hepatotoxicity 9%
• Xenobiotic Metabolism 9%
• Ethanol 9%