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CCL2 disrupts the adherens junction: Implications for neuroinflammation
Toni K. Roberts
,
Eliseo A. Eugenin
, Lillie Lopez
, Ignacio A. Romero
, Babette B. Weksler
, Pierre Olivier Couraud
, Joan W. Berman
Research output
:
Contribution to journal
›
Article
›
peer-review
95
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Scopus citations
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Keyphrases
Chemokine (C-C motif) Ligand 2 (CCL2)
100%
Neuroinflammation
100%
Adherens Junction
100%
PECAM-1
83%
Catenin
50%
Endothelial Dysfunction
16%
Leukocytes
16%
Phosphatase
16%
Endothelium
16%
Central Nervous System
16%
Blood-brain Barrier
16%
Tyrosine
16%
Human Brain
16%
Adhesion Molecules
16%
Protein Phosphatase
16%
Reassociation
16%
SH3 Domain
16%
Blood-brain Barrier Disruption
16%
Leukocyte Recruitment
16%
Endothelial Junctions
16%
Central Nervous System Pathology
16%
Central Nervous System Inflammation
16%
Brain Microvascular
16%
Chemokine CCL2
16%
SHP2
16%
Anti-neuroinflammation
16%
Surface Localization
16%
Biochemistry, Genetics and Molecular Biology
CCL2
100%
Adherens Junction
100%
Neuroinflammation
100%
Cell Adhesion Molecule 1
71%
Platelet
71%
Catenin
42%
Leukocyte
28%
Blood Brain Barrier
28%
Cell Adhesion Proteins
14%
Chemokine
14%
Tyrosine
14%
Phosphoprotein Phosphatase
14%
Phosphatase
14%
Medicine and Dentistry
Neuroinflammation
100%
Adherens Junction
100%
Platelet Endothelial Cell Adhesion Molecule 1
83%
Central Nervous System
50%
Catenin
50%
Blood Brain Barrier
33%
Endothelial Dysfunction
16%
Leukocyte
16%
Neuropathology
16%
Monocyte Chemotactic Protein 1
16%
Leukocyte Recruitment
16%
Phosphoprotein Phosphatase
16%
Protein Tyrosine Phosphatase SHP 2
16%
Cell Adhesion Proteins
16%
Diseases
16%
Immunology and Microbiology
CCL2
100%
Adherens Junction
100%
Platelet Endothelial Cell Adhesion Molecule 1
83%
Catenin
50%
Central Nervous System
50%
Blood Brain Barrier
33%
Phosphatase
33%
Leukocyte
16%
Monocyte Chemotactic Protein 1
16%
Leukocyte Recruitment
16%
Cell Adhesion Proteins
16%
Tyrosine
16%