Coagulation factors, fibrinogen and plasminogen activator inhibitor-1, are differentially regulated by yellow fever virus infection of hepatocytes

Keyphrases

• Virus Infection 100%
• Plasminogen Activator inhibitor-1 (PAI-1) 100%
• Yellow Fever 100%
• Hepatocytes 100%
• Coagulation Factors 100%
• Fibrinogen 100%
• Liver 40%
• Bleeding 40%
• Human Hepatocytes 40%
• Molecular Mechanism 20%
• Flavivirus 20%
• Age of Infection 20%
• Interleukin-6 20%
• South America 20%
• Dengue 20%
• Unvaccinated 20%
• Africa 20%
• Expression Level 20%
• Hemorrhagic Disease 20%
• Endemic Region 20%
• Disease Manifestations 20%
• Prothrombin Time 20%
• Coagulation Abnormalities 20%
• D-dimer 20%
• Fibrin Split Products 20%
• Clotting Factors 20%
• Overall Outcome 20%
• Coagulation Pathway 20%
• Hematemesis 20%
• Ecchymosis 20%
• Time Reduction 20%
• Clotting Time 20%
• Petechiae 20%

Medicine and Dentistry

• Infection 100%
• Virus Infection 100%
• Hepatocyte 100%
• Yellow Fever Virus 100%
• Blood Clotting Factor 100%
• Fibrinogen 100%
• Plasminogen Activator Inhibitor-1 100%
• Disease 40%
• Flavivirus 20%
• Bleeding 20%
• Dengue Fever 20%
• Interleukin 6 20%
• Prothrombin Time 20%
• Bleeding Disorder 20%
• Hematemesis 20%
• Fibrin Degradation Product 20%
• D-Dimer 20%
• Ecchymosis 20%
• Punctate Hemorrhage 20%

Pharmacology, Toxicology and Pharmaceutical Science

• Infection 100%
• Plasminogen Activator Inhibitor 1 100%
• Virus Infection 100%
• Yellow Fever Virus 100%
• Blood Clotting Factor 100%
• Fibrinogen 100%
• Disease 40%
• Bleeding 20%
• Flavivirus 20%
• Interleukin 6 20%
• Dengue 20%
• Prothrombin 20%
• D Dimer 20%
• Bleeding Disorder 20%
• Fibrin Degradation Product 20%
• Clotting Time 20%
• Hematemesis 20%
• Petechia 20%
• Ecchymosis 20%