Control of neuronal ion channel function by glycogen synthase kinase-3: New prospective for an old kinase

Keyphrases

• Addictive Behavior 9%
• Adulthood 9%
• Brain Diseases 9%
• Brain Plasticity 9%
• Central Nervous System 18%
• Cognitive Processing 9%
• Disease Intervention 9%
• Downstream Effectors 9%
• Emotional Processing 9%
• Evolutionarily Conserved 9%
• Excitability 9%
• Glutamate Receptors 9%
• Glycogen Synthase Kinase 3 (GSK3) 100%
• Human Disease 9%
• Intracellular Signaling 9%
• Ion Channel Function 100%
• Ion Channels 9%
• Ligand-gated 9%
• Maladaptive Plasticity 9%
• Medication Development 9%
• Molecular Targets 9%
• Na + 9%
• Neurodegenerative Diseases 9%
• Neuronal Circuitry 9%
• Neuronal Function 9%
• Neurotransmitter Release 9%
• New Evidence 9%
• Pharmacological Intervention 9%
• Phosphorylation 9%
• Promising Target 9%
• Psychiatric Disorders 9%
• Synaptic Transmission 9%
• Therapeutic Intervention 9%
• Voltage Gating 9%

Neuroscience

• Addictive Behavior 9%
• Brain Disease 9%
• Brain Plasticity 9%
• Central Nervous System 18%
• Excitability 9%
• Glutamate Receptor 9%
• Glycogen Synthase Kinase 3 100%
• Intracellular Signaling 9%
• Ion Channel 100%
• Kinase 100%
• Mental Disorder 9%
• Neurodegenerative Disorder 9%
• Neurotransmitter Release 9%
• Synaptic Transmission 9%