Abstract
Two rad mutants of yeast, rad10 and rad16, are shown to be defective in the removal of UV-induced pyrimidine dimers since DNAs obtained from irradiated cells following a post-irradiation incubation in the dark still retain UV-endonuclease-sensitive sites. Both rad10 and rad16 mutants are in the same pathway of excision-repair as the rad1, rad2, rad3 and rad4 mutants.
Original language | English (US) |
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Pages (from-to) | 125-128 |
Number of pages | 4 |
Journal | MGG Molecular & General Genetics |
Volume | 152 |
Issue number | 2 |
DOIs | |
State | Published - Jan 1977 |
Externally published | Yes |
ASJC Scopus subject areas
- Genetics