Differential modulation of base excision repair activities during brain ontogeny: Implications for repair of transcribed DNA

Keyphrases

• 5-hydroxymethyluracil 33%
• AP Endonuclease 33%
• APE1 66%
• Base Excision Repair 100%
• Base Excision Repair Enzymes 66%
• Base Excision Repair Pathway 33%
• Brain Development 33%
• Brain Ontogeny 100%
• Differential Modulation 100%
• DNA Glycosylase 66%
• DNA Repair 33%
• Excise 33%
• Excision 66%
• Glycoside Hydrolase 66%
• High-flow Oxygen 33%
• Independent Basis 33%
• Mammalian Cells 33%
• Mature Rats 33%
• NEIL1 66%
• NEIL2 33%
• Oxidative DNA Damage 66%
• Oxidized Bases 33%
• Oxygen Consumption 33%
• Phosphate 33%
• Physiologic 33%
• Post-mitotic 100%
• Postmitotic Neurons 33%
• Proliferating Cells 33%
• Rat Brain 33%
• Repair Method 100%
• Subpath 33%

Biochemistry, Genetics and Molecular Biology

• AP Endonuclease 20%
• Base Excision Repair 100%
• Brain Development 20%
• Cell Proliferation 20%
• DNA Damage 40%
• DNA Glycosylase 40%
• DNA Repair 20%
• Genomics 20%
• Glycosylase 40%
• Mammal 20%
• Mammalian Cell 20%
• NEIL1 40%
• Ontogeny 100%
• Oxygen Consumption 20%
• Repair Enzyme 40%