Dissemination of non-typhoidal Salmonella during Plasmodium chabaudi infection affects anti-malarial immunity

Keyphrases

• Plasmodium Chabaudi 100%
• Non-typhoidal Salmonella 100%
• Malaria 66%
• Chabaudi 66%
• Bacteremia 50%
• Plasmodium Infection 50%
• Adaptive Immune Response 33%
• Infected Mice 33%
• Intestinal Barrier Function 33%
• Intestine 16%
• Liver 16%
• Bacterial Translocation 16%
• Flow Cytometry 16%
• Gut Bacterial Translocation 16%
• Specific Immune Response 16%
• T Cell Activation 16%
• High Mortality Rate 16%
• Major Complications 16%
• Gastrointestinal Pathogens 16%
• Salmonella Infection 16%
• Barrier Function 16%
• Intestinal Wall 16%
• Cell Infiltration 16%
• Lipopolysaccharide-binding Protein 16%
• Light Microscopy 16%
• Large Molecules 16%
• Severe Malaria 16%
• Intestinal Tract 16%
• B Cell Activation 16%
• Soluble Substances 16%
• Intestinal Translocation 16%
• Intestinal Histology 16%
• Salmonella Bacteremia 16%
• Malarial Infection 16%

Pharmacology, Toxicology and Pharmaceutical Science

• Salmonella 100%
• Antimalarial Agent 100%
• Plasmodium Chabaudi Infection 100%
• Infection 66%
• Bloodstream Infection 66%
• Plasmodium 50%
• Wound 16%
• Infectious Agent 16%
• Mortality Rate 16%
• Flow Cytometry 16%
• Salmonellosis 16%
• Lipopolysaccharide Binding Protein 16%

Immunology and Microbiology

• Salmonella 100%
• Plasmodium chabaudi 100%
• Antimalarial Agent 100%
• Plasmodium 42%
• Bacterial Translocation 28%
• Adaptive Immune System 28%
• Immune Response 14%
• Infectious Agent 14%
• Intestine 14%
• Mortality Rate 14%
• T Cell Activation 14%
• Light Microscopy 14%
• B Cell Activation 14%
• Lipopolysaccharide Binding Protein 14%