Endothelial glutathione-S-transferase A4-4 protects against oxidative stress and modulates iNOS expression through NF-κB translocation

Keyphrases

• 4-hydroxynonenal (4-HNE) 100%
• Advanced Plaque 10%
• Aldehydes 10%
• Atherogenesis 10%
• Atherosclerotic Plaque 10%
• Conflicting Results 10%
• Defensive Response 10%
• Detoxification 20%
• Early Humans 10%
• Endothelial Cells 10%
• Endothelium 10%
• Enzymatic Defense System 10%
• Gene Expression 10%
• Glutathione S-transferase 20%
• Glutathione S-transferase A4 100%
• GSTA4 20%
• High Activity 10%
• Human Plaque 10%
• Immune Response 10%
• Immunohistochemical Study 10%
• Induced Apoptosis 10%
• Inducible Genes 10%
• Inducible Nitric Oxide Synthase 100%
• Inflammation Response 10%
• Mouse Pancreatic Islets 10%
• Nitric Oxide 10%
• Nuclear factor-B 100%
• Overexpression 10%
• Oxidative Damage 10%
• Oxidative Stress 100%
• RB Pathway 10%
• Transfected Cells 20%

Pharmacology, Toxicology and Pharmaceutical Science

• Alternative Complement Pathway C3 C5 Convertase 100%
• Atherogenesis 16%
• Detoxication 33%
• Glutathione 33%
• Glutathione Transferase A4 100%
• Inducible Nitric Oxide Synthase 100%
• Inflammation 16%
• Nitric Oxide 16%
• Nuclear Factor 100%