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Epstein-Barr virus infection of Langerhans cell precursors as a mechanism of oral epithelial entry, persistence, and reactivation
Dennis M. Walling
, Autumn J. Ray
, Joan E. Nichols
, Catherine M. Flaitz
, C. Mark Nichols
Research output
:
Contribution to journal
›
Article
›
peer-review
45
Scopus citations
Overview
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Dive into the research topics of 'Epstein-Barr virus infection of Langerhans cell precursors as a mechanism of oral epithelial entry, persistence, and reactivation'. Together they form a unique fingerprint.
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Medicine and Dentistry
Precursor
100%
Epstein Barr Virus
100%
Langerhans Cell
100%
Epstein-Barr Virus Infection
100%
Oral
100%
Diseases
20%
Infection
10%
Herpesviridae
10%
B Lymphocyte
10%
Mononuclear Cell
10%
Persistent Virus Infection
10%
Keyphrases
Langerhans Cells
100%
Epstein-Barr Virus
100%
Epstein-Barr Virus Infections
100%
Cell Precursors
100%
Oral Epithelium
30%
Blood-borne
20%
Human Disease
10%
Natural History
10%
B Lymphocytes
10%
Mononuclear Cells
10%
Nonmalignant
10%
Virus Persistence
10%
Resident Cells
10%
Mucosal Epithelium
10%
Human Herpesvirus
10%
Infectious Reservoir
10%
Immunology and Microbiology
Precursor
100%
Viral Disease
100%
Langerhans Cell
100%
Epstein Barr Virus
100%
B Cell
8%
Mononuclear Cell
8%
Herpesviridae
8%
Infection
8%