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Exogenous Bcl-X
L
fusion protein spares neurons after spinal cord injury
Olivera Nesic-Taylor
, D. Cittelly
, Z. Ye
, G. Y. Xu
, G. Unabia
, J. C. Lee
, N. M. Svrakic
, X. H. Liu
, R. J. Youle
, T. G. Wood
, D. McAdoo
, K. N. Westlund
, C. E. Hulsebosch
, J. R. Perez-Polo
Biochemistry & Molecular Biology
Research output
:
Contribution to journal
›
Article
›
peer-review
72
Scopus citations
Overview
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L
fusion protein spares neurons after spinal cord injury'. Together they form a unique fingerprint.
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Keyphrases
Bcl-xL
100%
Spinal Cord Injury
100%
Fusion Protein
100%
Pro-apoptotic
23%
Neuronal Death
23%
Apoptosis
7%
Spinal Cord
7%
Dorsal Horn Neurons
7%
Mitochondria
7%
Motor Function
7%
Neuron Cells
7%
Clinically Significant
7%
Central Nervous System
7%
Anti-apoptotic
7%
DNA Microarray
7%
Protein Level
7%
B-cell Lymphoma 2 (Bcl-2)
7%
Motoneuron
7%
Sensory Function
7%
Neuronal Survival
7%
Affymetrix
7%
Ventral Horn
7%
Cellular Compartments
7%
Anti-apoptotic Bcl-2
7%
Transcriptional Changes
7%
Bcl-2 Family Proteins
7%
Injured Spinal Cord
7%
Glia Cells
7%
Bcl-w
7%
Mitochondrium
7%
Neuroscience
Spinal Cord Injury
100%
Chimeric Protein
100%
Neuronal Cell Death
50%
Bcl-2
33%
Glia
16%
Central Nervous System
16%
Mitochondrion
16%
DNA Microarray
16%
Protein Bcl Xl
16%
Programmed Cell Death
16%