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Hypoaldosteronism — Disease or Normal Response?
O. Bryan Holland
Internal Medicine
Research output
:
Contribution to journal
›
Editorial
›
peer-review
9
Scopus citations
Overview
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Biochemistry, Genetics and Molecular Biology
ATPase
100%
Secretion (Process)
100%
Basolateral Membrane
100%
Glucocorticoid
100%
Kidney Function
100%
Aldosterone Release
100%
Aldosterone
100%
Aldosterone Urine Level
100%
Hyperkalemia
100%
Potassium Excretion
100%
Kidney Tubule Absorption
100%
Permease
100%
Keyphrases
Hyperaldosteronism
100%
Aldosterone
100%
Disease Response
100%
Normal Response
100%
ATP Synthase
50%
Basolateral Membrane
50%
Renal Tubular Epithelial Cells
50%
Sodium
50%
Aldosterone Secretion
50%
Na +
50%
Hyperkalemia
50%
Sodium-to-potassium Ratio
50%
Permease
50%
Normal Renal Function
50%
Luminal Membrane
50%
Isolated Hypoaldosteronism
50%
Renal Reabsorption
50%
Potassium Excretion
50%