Ifn-γ drives tnf-α hyperproduction and lethal lung inflammation during antibiotic treatment of postinfluenza staphylococcus aureus pneumonia

Keyphrases

• Lung Damage 100%
• Lung Inflammation 100%
• Antibiotic Treatment 100%
• Streptococcus Pneumoniae 100%
• Hyperproduction 100%
• Antibiotics 66%
• Methicillin-resistant Staphylococcus Aureus Pneumonia 66%
• Influenza 66%
• Antibiotic Therapy 66%
• Acute Respiratory Distress Syndrome 66%
• Inflammatory Cytokines 33%
• Mouse Model 33%
• Methicillin-resistant Staphylococcus Aureus 33%
• Cytokine Response 33%
• Bacterial Clearance 33%
• Inflammatory Cytokine Response 33%
• Superinfection 33%
• Syndrome Model 33%
• Clinical Scenarios 33%
• IFN Response 33%
• Animal Mortality 33%
• Effective Rate of Protection 33%
• Bacterial Control 33%
• Inflammatory Cytokine Storm 33%

Pharmacology, Toxicology and Pharmaceutical Science

• Antibiotics 100%
• Lung Injury 100%
• Pneumonia 100%
• Staphylococcal Pneumonia 100%
• Antibiotic Therapy 66%
• Cytokine 66%
• Antibiotic Agent 66%
• Methicillin Resistant Staphylococcus Aureus Infection 66%
• Adult Respiratory Distress Syndrome 66%
• Mouse 33%
• Mouse Model 33%
• Tumor Necrosis Factor 33%
• Methicillin-Resistant Staphylococcus Aureus 33%
• Cytokine Storm 33%
• Bacterial Clearance 33%
• Superinfection 33%

Immunology and Microbiology

• Pulmonary Inflammation 100%
• Methicillin-Resistant Staphylococcus Aureus 100%
• Staphylococcus Aureus 100%
• Influenza 66%
• Inflammatory Cytokine 66%
• Cytokine Response 66%
• Proinflammatory Cytokine 33%
• Mouse 33%
• Wild Type 33%
• Mouse Model 33%
• Tumor Necrosis Factor 33%
• Neutralization 33%
• Cytokine Storm 33%
• Bacterial Clearance 33%
• Superinfection 33%