Immunopathogenesis of non-healing American cutaneous leishmaniasis and progressive visceral leishmaniasis

Keyphrases

• American Cutaneous Leishmaniasis 100%
• Non-healing 100%
• Visceral Leishmaniasis 100%
• Immunopathogenesis 100%
• T Cell Response 33%
• Immune Response 33%
• Leishmania 33%
• Leishmania Infection 33%
• Leishmania Braziliensis 33%
• Leishmania Amazonensis 33%
• Arginase 33%
• Leishmania Donovani 33%
• Cutaneous Leishmaniasis 33%
• Cell Activation 33%
• Cell Signaling Pathways 33%
• Th1-Th2 33%
• Host Immune Response 33%
• Dysregulated 33%
• Host Susceptibility 33%
• Macrophage Activation 33%
• Co-infection 33%
• Th2 Cells 33%
• Infection Control 33%
• Interleukin-18 (IL-18) 33%
• Parasite Replication 33%
• Th1 Cells 33%
• Interleukin-17 (IL-17) 33%
• Protective Response 33%
• Subclinical Infection 33%
• Parasite Growth 33%
• Regulatory T Cells 33%
• Self-healing 33%
• Hyper-response 33%
• Interleukin-27 (IL-27) 33%
• Th1 Differentiation 33%
• Parasite Species 33%
• Effector T Cell Response 33%
• IL-17-producing T Cells 33%
• Local Lesions 33%
• Nutrition Status 33%
• Innate Immunity Cells 33%
• Generalized Consensus 33%
• Rational Control 33%
• Host nutrition 33%
• Innate-like T Cells 33%

Immunology and Microbiology

• Cutaneous Leishmaniasis 100%
• Visceral Leishmaniasis 100%
• Immunopathogenesis 100%
• T Cell 66%
• Signal Transduction 66%
• Cytokine 66%
• Interleukin 17 66%
• Innate Immune System 33%
• Animal Experiment 33%
• Leishmaniasis 33%
• Leishmania 33%
• Infectious Agent 33%
• Macrophage Activation 33%
• Cell Activation 33%
• Regulatory T Cell 33%
• Host Susceptibility 33%
• Coinfection 33%
• Th2 Cell 33%
• Interleukin 18 33%
• Leishmania amazonensis 33%
• Leishmania donovani 33%
• Th1 Cell 33%
• Leishmania braziliensis 33%