Lack of PD-L1 Expression by iNKT Cells Improves the Course of Influenza A Infection

Keyphrases

• PD-L1 Expression 100%
• Influenza-like Illness 100%
• Invariant Natural Killer T Cells 100%
• Programmed Death-ligand 1 (PD-L1) 60%
• PD-L2 50%
• Gene-deficient Mice 40%
• Influenza Virus 40%
• Influenza 30%
• Pulmonary Inflammation 20%
• Weight Loss 20%
• Cell Infiltration 20%
• Derived Invariants 20%
• Inflammatory Response 10%
• Wild Mice 10%
• Interleukin-4 (IL-4) 10%
• Interleukin-13 (IL-13) 10%
• T Cell Subsets 10%
• Lung 10%
• New Therapies 10%
• Transfer System 10%
• Infected Mice 10%
• Inhibitory Receptors 10%
• Costimulatory Molecules 10%
• Interferon-γ 10%
• Adoptive Transfer 10%
• L1-L2 10%
• Negatively Regulate 10%
• Reduced Sensitivity 10%

Immunology and Microbiology

• Influenza 100%
• Programmed Death-Ligand 1 100%
• Natural Killer T Cell 100%
• Infection 100%
• Influenzavirus A 60%
• Viral Disease 20%
• Wild Type 20%
• Pulmonary Inflammation 20%
• Wild Type Mouse 10%
• Inflammation Response 10%
• Cytokine 10%
• Interleukin 13 10%
• T Cell Subset 10%
• Interleukin 4 10%
• Co-Stimulatory Molecule 10%
• Interferon Gamma 10%
• Programmed Death 1 Ligand 1 10%
• Programmed Death 1 Receptor 10%
• Programmed Death 1 Ligand 2 10%

Biochemistry, Genetics and Molecular Biology

• Natural Killer T Cell 100%
• PD-L1 100%
• Influenza A Virus 60%
• Wild Type 20%
• Cytokine 10%
• Wild Type Mouse 10%
• Interferon Gamma 10%
• Interleukin 13 10%
• Interleukin 4 10%