Mechanisms of intracellular killing of Rickettsia conorii in infected human endothelial cells, hepatocytes, and macrophages

Keyphrases

• Chemokines 60%
• Controversial Issues 20%
• Degradative Enzymes 20%
• Endothelium 40%
• Hepatocytes 100%
• Host Cell 20%
• Human Cells 40%
• Human Endothelial Cells 100%
• Human Hepatocytes 40%
• Human Macrophages 40%
• Human Peripheral Blood Monocytes 20%
• Human Target 20%
• Human Umbilical Vein Endothelial Cells (HUVECs) 40%
• Human-to-human 20%
• Hydrogen Peroxide 20%
• Hydrogen Peroxide Production 20%
• Indoleamine 2,3-dioxygenase 1 (IDO1) 20%
• Interferon-γ 20%
• Interleukin-1β 20%
• Intracellular Killing 100%
• Intracellular Pathogen 20%
• Macrophage Cell Line 20%
• Macrophages 100%
• Nitric Oxide 100%
• Nitric Oxide Production 20%
• Rickettsia 60%
• Rickettsia Conorii 100%
• Rickettsial 20%
• Rickettsial Infection 40%
• T Cells 60%
• THP-1 Cells 20%
• Tryptophan 40%
• Tryptophan Degradation 20%
• Tumor Necrosis Factor-α 20%

Immunology and Microbiology

• Chemokine 75%
• Cytokine 50%
• Host Cell 25%
• Human Cell 100%
• Hydrogen Peroxide 50%
• Indoleamine 2,3-dioxygenase 25%
• Infectious Agent 25%
• Interferon Gamma 25%
• Interleukin 1 25%
• Intracellular Killing 100%
• Macrophage 100%
• Macrophage Cell Line 25%
• Rickettsia 75%
• Rickettsia conorii 100%
• T Cell 75%
• Target Cell 25%
• Tumor Necrosis Factor Alpha 25%
• Umbilical Vein Endothelial Cell 50%