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Mitochondrial Ca
2+
uptake is essential for synaptic plasticity in pain
Hee Young Kim
, Kwan Yeop Lee
, Ying Lu
, Jigong Wang
, Lian Cui
, Sang Jeong Kim
, Jin Mo Chung
, Kyungsoon Chung
Research output
:
Contribution to journal
›
Article
›
peer-review
79
Scopus citations
Overview
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Dive into the research topics of 'Mitochondrial Ca
2+
uptake is essential for synaptic plasticity in pain'. Together they form a unique fingerprint.
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Keyphrases
Synaptic Plasticity
100%
Ca2+
100%
Mitochondrial Ca2+ Uptake
100%
Long-term Potentiation
50%
Hyperalgesia
50%
N-methyl-D-aspartate Receptor (NMDAR)
25%
Reactive Oxygen Species
25%
Spinal Cord
25%
Mitochondria
25%
Cell Signaling Pathways
25%
Persistent Pain
25%
Reactive Oxygen Species Production
25%
Chronic Pain
25%
Receptor Activation
25%
Chronic Pain Treatment
25%
Cell Signaling
25%
Cytosolic Free Ca2+
25%
Mitochondrial Superoxide
25%
Mitochondrium
25%
Medicine and Dentistry
Calcium Ion
100%
Nerve Cell Plasticity
100%
Reactive Oxygen Species
22%
Chronic Pain
22%
Hyperpathia
22%
Long-Term Potentiation
22%
Signal Transduction
11%
Persistent Pain
11%
Superoxide
11%
Mitochondrion
11%
Signal Transduction Pathway
11%
Biological Product
11%
NMDA Receptor
11%