Mixtures of xenoestrogens disrupt estradiol-induced non-genomic signaling and downstream functions in pituitary cells

Keyphrases

• Xeno-oestrogens 100%
• Non-canonical Pathway 100%
• Estradiol 100%
• Pituitary Cells 100%
• Bisphenol A 60%
• 4-Nitrophenol 30%
• C-Jun N-terminal Kinase 30%
• Physiologic 20%
• Estrogen 20%
• Functional Response 20%
• Extracellular Signal-regulated Kinase 20%
• Caspase-8 20%
• Apoptosis 10%
• Dose-response 10%
• Intracellular Signaling 10%
• Cellular Response 10%
• Phosphorylation 10%
• Prolactin 10%
• Induced Response 10%
• Estrogenic 10%
• Prolactin Release 10%
• Radioimmunoassay 10%
• Amino 10%
• Cell Proliferation 10%
• Cell number 10%
• Environmental Concentrations 10%
• Proliferative Effect 10%
• Apoptotic Response 10%
• Cell Regulation 10%
• Rat pituitary 10%
• Estrogenic Response 10%
• Immunoassay 10%
• Vehicle Control 10%
• Surfactant 10%
• Contaminated Environment 10%
• Consumer Products 10%
• Caspase-9 10%
• Fluorescent Compounds 10%
• Caspase-9 Activity 10%
• Pituitary Cell Line 10%
• Crystal Violet Assay 10%
• Hormonal Pathway 10%
• Bisphenol Compounds 10%
• Medium Throughput 10%
• Mixture Effects 10%

Pharmacology, Toxicology and Pharmaceutical Science

• Diethylstilbestrol 100%
• Xenoestrogen 100%
• Nonylphenol 30%
• 4,4' Isopropylidenediphenol 30%
• Immunoassay 20%
• Mitogen Activated Protein Kinase 20%
• Prolactin 20%
• Caspase 8 20%
• Caspase 9 20%
• Surfactant 10%
• Crystal Violet 10%