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Nicotinic acetylcholine receptor interaction with β-amyloid: Molecular, cellular, and physiological consequences
H. R. Parri, K. T. Dineley
Neurology
Research output
:
Contribution to journal
›
Review article
›
peer-review
63
Scopus citations
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Dive into the research topics of 'Nicotinic acetylcholine receptor interaction with β-amyloid: Molecular, cellular, and physiological consequences'. Together they form a unique fingerprint.
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Keyphrases
Alzheimer's Disease
40%
Amyloid-β Peptide
20%
Cellular Level
20%
Etiology
40%
Molecular Consequences
100%
Nicotinic Acetylcholine Receptor (nAChR)
100%
Physiological Consequences
100%
Physiological Level
20%
Physiological Processes
20%
Receptor-ligand Interaction
100%
Medicine and Dentistry
Amyloid Protein
100%
Etiology of Alzheimer's Disease
66%
Nicotinic Receptor
100%
Physiological Process
33%
Biochemistry, Genetics and Molecular Biology
Amyloid Protein
100%
Nicotinic Acetylcholine Receptor
100%
Physiological Process
33%
Agricultural and Biological Sciences
Amyloid Protein
100%
Cholinergic Receptor
100%
Chemistry
Acetylcholine
100%