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Pathogenesis of ross river virus infection in mice. I.ependymal infection, cortical thinning, and hydrocephalus
C. A. Mims, F. A. Murphy
Research output
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Contribution to journal
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Article
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peer-review
60
Scopus citations
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Dive into the research topics of 'Pathogenesis of ross river virus infection in mice. I.ependymal infection, cortical thinning, and hydrocephalus'. Together they form a unique fingerprint.
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Keyphrases
Virus Infection
100%
Hydrocephalus
100%
Ependymal
100%
Ross River Virus
100%
Cortical Thinning
100%
Necrosis
75%
Aged Mice
50%
Central Nervous System
50%
Cerebral Cortex
50%
Paralysis
50%
Retina
25%
Pathological Changes
25%
Apparently Healthy
25%
Acute Infection
25%
Semliki Forest Virus
25%
Infection Focus
25%
Ependyma
25%
Extraneural
25%
Cortical Lesions
25%
Aqueductal Stenosis
25%
Healthy Mice
25%
Ependymal Cells
25%
Fluid-filled
25%
Neuroscience
Hydrocephalus
100%
Ross River Virus
100%
Necrosis
75%
Central Nervous System
50%
Cerebral Cortex
50%
Stenosis
25%
Ependyma
25%
Semliki Forest Virus
25%
Ependymal Cell
25%
Veterinary Science and Veterinary Medicine
Mouse
100%
Central Nervous System
33%
Ependyma
16%
Aqueductal Stenosis
16%