Perinatal deiodinase 2 expression in hepatocytes defines epigenetic susceptibility to liver steatosis and obesity

Keyphrases

• Obesity 100%
• Epigenetics 100%
• Hepatocytes 100%
• Liver Steatosis 100%
• High-fat Diet 100%
• Type 2 Deiodinase 100%
• Fatty Acids 50%
• Hypertriglyceridemia 50%
• Lipid Metabolism 50%
• Gene Transcription 50%
• Transcriptional Response 50%
• Steatosis 50%
• Prohormone 50%
• Receptor Expression 50%
• Triglycerides 50%
• Chromatin 50%
• Thyroid Hormones 50%
• Peroxisome Proliferator-activated Receptor 50%
• Diet-induced 50%
• Nuclear Receptor 50%
• Triiodothyronine 50%
• Lipid Metabolism Genes 50%
• Transport Activity 50%
• First Day 50%
• Epigenetic Modification 50%
• Thyroxine 50%
• Reduced Susceptibility 50%
• Hepatocyte Function 50%
• Analytical Platforms 50%
• Methylation Profile 50%
• Reverse Cholesterol Transport 50%
• Hepatocyte-specific 50%
• Whole-genome Methylation 50%
• Lipase Activity 50%
• Local Region 50%
• Transient Surges 50%

Biochemistry, Genetics and Molecular Biology

• Epigenetics 100%
• Hepatocyte 100%
• Deiodinase 100%
• Lipid 33%
• Methylation 33%
• Genetic Transcription 33%
• Hypertriglyceridemia 33%
• Triglyceride 33%
• Liver Function 33%
• Lipid Metabolism 33%
• Triiodothyronine 33%
• Liothyronine 33%
• Nuclear Receptor 33%
• Peroxisome Proliferator-Activated Receptor 33%
• Epigenetic Modification 33%
• Triacylglycerol Lipase 33%
• Pancreatic Lipase 33%
• Reverse Cholesterol Transport 33%
• Thyroid Hormone 33%