Post-ischaemic thyroid hormone treatment in a rat model of acute stroke

Keyphrases

• Rat Model 100%
• Reperfusion 100%
• Transient Middle Cerebral Artery Occlusion (tMCAO) 100%
• Thyroid Hormone Replacement 100%
• Acute Stroke 100%
• Inducible Nitric Oxide Synthase 50%
• Cyclooxygenase-2 50%
• Transcription Factor 50%
• Ischemia 50%
• Ischemic Injury 50%
• Phosphorylation 50%
• Inflammatory Cells 50%
• Disability 50%
• Prohormone 50%
• Therapeutic Effect 50%
• Limited Treatment Options 50%
• Cellular Factors 50%
• Brain Injury 50%
• Anti-apoptotic 50%
• Neuroprotective Effect 50%
• Astrocyte Activation 50%
• Ischemic Brain 50%
• Cerebral Infarction 50%
• Focal Cerebral Ischemia 50%
• Decreased Expression 50%
• Rat Brain 50%
• Microglial Activation 50%
• Thyroxine 50%
• Infarct Volume 50%
• Neurotrophic Factors 50%
• Bcl-2 Family Proteins 50%
• Cerebral Infarct 50%
• Infarct Area 50%
• Anti-inflammatory Mechanism 50%
• Ischemic Stroke Treatment 50%
• Devastating Brain Injury 50%
• Promising Therapeutics 50%
• Neurotrophin Factors 50%

Neuroscience

• Brain Injury 100%
• Brain Ischemia 100%
• Middle Cerebral Artery 100%
• Hormone Therapy 100%
• Cerebrovascular Accident 100%
• Thyroid Hormone 100%
• Brain-Derived Neurotrophic Factor 50%
• Neurotrophin 50%
• Prehormone 50%
• Anti-Inflammatory 50%
• Neuroprotective Agent 50%
• Astrocyte 50%
• Enzymes 50%
• Cerebral Infarction 50%
• Microglia 50%
• Protein Bcl 2 50%
• Thyroxine 50%
• Neurotrophic Factor 50%
• Glial Cell Line-Derived Neurotrophic Factor 50%
• Ischemia 50%
• phosphorylation 50%