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Ral activation promotes melanomagenesis
P. A. Zipfel
, D. C. Brady
, D. F. Kashatus
, B. D. Ancrile
, D. S. Tyler
, C. M. Counter
Research output
:
Contribution to journal
›
Article
›
peer-review
41
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Scopus citations
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Keyphrases
Bound States
25%
BRAF mutation
25%
Cancer Cell Lines
25%
Effector Pathways
25%
Genotype
25%
Guanine Exchange Factor
75%
Guanosine Deaminase
25%
Guanosine Triphosphate
25%
Human Melanoma
50%
Knockdown
25%
Melanoma
100%
Melanoma Cells
50%
Melanomagenesis
100%
Negative Regulator
25%
NRAS mutation
25%
Oncogenic mutations
25%
Oncogenic Nras
25%
Phosphoinositide 3-kinase (PI3K)
75%
Primary Class
25%
RalA
75%
RalB
50%
Ras Activation
25%
Ras Effectors
50%
Ras Proteins
25%
RNA-based
25%
Short Hairpin RNA (shRNA)
25%
Small GTPase
25%
Tumorigenesis
25%
Medicine and Dentistry
Allele
50%
Cancer Cell Line
16%
Carcinogenesis
100%
Guanine
50%
Guanosine Triphosphatase
16%
Guanosine Triphosphate
16%
Melanoma
100%
Melanoma Cell Line
16%
Phosphoinositide 3-Kinase
33%
Ras Protein
16%
Ras Signaling
50%
Short Hairpin RNA
16%
Small GTPase
16%
Biochemistry, Genetics and Molecular Biology
Allele
50%
Cancer Cell
16%
Carcinogenesis
100%
GTPase
16%
Guanine
50%
Guanosine Triphosphate
16%
Phosphoinositide 3-Kinase
33%
Small GTPase
16%
Small Hairpin RNA
16%
Small Interfering RNA
16%
Wild Type
16%