Regulation and role of endogenously produced hydrogen sulfide in angiogenesis

Keyphrases

• Angiogenesis 100%
• Hydrogen Sulfide 100%
• Vascular Endothelial Growth Factor 66%
• Wound Healing 33%
• Angiogenic Factors 33%
• Overexpression 33%
• Signaling Pathway 33%
• Therapeutic Potential 33%
• Endothelial Cells 33%
• Mitogen-activated Protein Kinase 33%
• Novel Therapeutics 33%
• Pharmacological Inhibitors 33%
• Nitric Oxide 33%
• Phosphoinositide 3-kinase (PI3K) 33%
• Proliferation Migration 33%
• Cyclic GMP 33%
• Cultured Endothelial Cells 33%
• H2S-producing Enzymes 33%
• Neovascularization 33%
• Enhanced Production 33%
• Angiogenic 33%
• ATP-sensitive Potassium Channel 33%
• Angiogenic Response 33%
• Tumor Angiogenesis 33%
• Angiogenic Property 33%
• Angiogenic Signaling 33%
• Network Formation 33%
• Post-ischemic Angiogenesis 33%
• Regulated Cascade 33%
• Prototypical Model 33%
• Tube Type 33%
• Substrate Supplementation 33%
• Structured Networks 33%

Pharmacology, Toxicology and Pharmaceutical Science

• Vasculotropin 100%
• Hydrogen Sulfide 100%
• Wound Healing 50%
• Disease 50%
• Neoplasm 50%
• Nitric Oxide 50%
• Mitogen Activated Protein Kinase 50%
• Phosphatidylinositol 3 Kinase 50%
• Neovascularization (Pathology) 50%
• Angiogenic Factor 50%
• Adenosine Triphosphate Sensitive Potassium Channel 50%

Biochemistry, Genetics and Molecular Biology

• Angiogenesis 100%
• Hydrogen Sulfide 100%
• Enzyme 60%
• Vascular Endothelial Growth Factor 40%
• Wound Healing 20%
• Signal Transduction 20%
• Nitric Oxide 20%
• Mitogen-Activated Protein Kinase 20%
• Phosphoinositide 3-Kinase 20%
• Neovascularization (Pathology) 20%
• Adenosine Triphosphate Sensitive Potassium Channel 20%