Respiratory syncytial virus infection down-regulates antioxidant enzyme expression by triggering deacetylation-proteasomal degradation of Nrf2

Keyphrases

• Respiratory Syncytial Virus Infection 100%
• Nuclear Factor Erythroid 2-related Factor 2 (Nrf2) 100%
• Proteasomal Degradation 100%
• Antioxidant Gene Expression 100%
• Deacetylation 100%
• Respiratory Syncytial Virus 50%
• Virus 25%
• Oxidative Stress 25%
• Airway 25%
• Antioxidant Enzymes 25%
• Inducer 12%
• Inflammation 12%
• Therapeutic Intervention 12%
• Transcription Factor 12%
• Lung Damage 12%
• Lung Disease 12%
• Reactive Oxygen Species Production 12%
• Viral 12%
• Hospitalization 12%
• Clinical Disease 12%
• Antioxidant 12%
• Protease Inhibitors 12%
• Rapid Generation 12%
• Oxidative Stress Damage 12%
• Cellular Level 12%
• Acute Respiratory Tract Infection 12%
• Ubiquitin-proteasome System 12%
• Gene Promoter 12%
• Antioxidant Capacity 12%
• Histone Deacetylase Inhibitor (HDACi) 12%
• Child-led 12%
• Progressive Reduction 12%
• Antioxidant Genes 12%
• Endogenous Promoter 12%
• Endogenous Antioxidant Enzymes 12%

Biochemistry, Genetics and Molecular Biology

• Protein Expression 100%
• Human Respiratory Syncytial Virus 100%
• Antioxidant Enzyme 100%
• Transcription Factor Nrf2 100%
• Deacetylation 100%
• Oxidative Stress 33%
• Promoter Region 11%
• Proteasome 11%
• Gene Promoter 11%
• Histone Deacetylase 11%
• Proteasome Inhibitor 11%

Immunology and Microbiology

• Respiratory Syncytial Virus Infection 100%
• Transcription Factor Nrf2 100%
• Protein Expression 100%
• Deacetylation 100%
• Human Respiratory Syncytial Virus 50%
• Oxidative Stress 37%
• Promoter Region 25%
• Proteasome 25%