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Role of polymorphic GSTM1 and GSTT1 genotypes on NNK-induced genotoxicity
S. A. Salama
, S. Z. Abdel-Rahman
, C. H. Sierra-Torres
, F. A. Hamada
, William W. Au
Obstetrics & Gynecology
Research output
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Contribution to journal
›
Article
›
peer-review
35
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Scopus citations
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Keyphrases
GSTT1
100%
GSTM1
100%
Genotype
100%
Genotoxicity
100%
Chromosomal Aberrations
40%
Polymorphism
30%
Concentration-dependent
20%
Sister Chromatid Exchange
20%
Chromosomal Aberration Test
20%
Null Cell
10%
Health Effects
10%
No Significant Difference
10%
Fluorescence in Situ Hybridization
10%
4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone
10%
N-nitrosamines
10%
Dominant Effect
10%
Glutathione Ethyl Ester
10%
Environmental Carcinogenesis
10%
GSTM1 null
10%
Tobacco-related
10%
Agricultural and Biological Sciences
Chromosome Aberration
100%
Genotoxicity
100%
Genotyping
66%
Polymorphism
50%
Sisters
33%
Chromatid
33%
Fluorescence in Situ Hybridization
16%
Null Cell
16%
Glutathione
16%
Malignant Neoplasm
16%
Pharmacology, Toxicology and Pharmaceutical Science
Nicotine-Derived Nitrosamine Ketone
100%
Genotoxicity
100%
Chromosome Aberration
66%
Malignant Neoplasm
11%
4 (Methylnitrosamino) 1 (3 Pyridyl) 1 Butanone
11%
Tobacco-Specific Nitrosamines
11%
Glutathione Ethyl Ester
11%
Veterinary Science and Veterinary Medicine
Cancer
100%
Glutathione
100%
In Situ Hybridization
100%
Food Science
Glutathione
100%