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Senescence of primary amniotic cells via oxidative DNA damage
Ramkumar Menon
, Istvan Boldogh
, Rheanna Urrabaz-Garza
, Jossimara Polettini
, Tariq Ali Syed
, George R. Saade
, John Papaconstantinou
, Robert N. Taylor
Obstetrics & Gynecology
Microbiology And Immunology
Biochemistry & Molecular Biology
Research output
:
Contribution to journal
›
Article
›
peer-review
105
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Scopus citations
Overview
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Dive into the research topics of 'Senescence of primary amniotic cells via oxidative DNA damage'. Together they form a unique fingerprint.
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Medicine and Dentistry
DNA Damage
100%
Amniocyte
100%
Reactive Oxygen Species
100%
Cigarette Smoke
83%
Premature Labor
50%
p38 Mitogen-Activated Protein Kinase
50%
Acetic Acid Derivative
33%
Antioxidant
33%
DNA Base
33%
DNA Strand
33%
Cysteine
33%
Repair Enzyme
33%
Enzyme Assay
33%
Oxidative Stress
16%
Programmed Cell Death
16%
Cell Culture
16%
Epithelial Cell
16%
Western Blot
16%
Phosphotransferase
16%
Complication of Pregnancy
16%
Fetal Membrane
16%
Amnion
16%
Comet Assay
16%
Fluorescein
16%
Cellular Senescence
16%
Etiology
16%
Post-Translational Modification
16%
phosphorylation
16%
Biochemistry, Genetics and Molecular Biology
DNA Damage
100%
Amnion Cell
100%
ASK1
75%
P38 Mitogen-Activated Protein Kinases
75%
Repair Enzyme
50%
Enzyme Assay
50%
Cysteine
50%
Cell Culture
25%
Oxidative Stress
25%
Posttranslational Modification
25%
Phosphotransferase
25%
Kinase
25%
DNA Strand
25%
Western Blot
25%
Fluorescein
25%
Amnion
25%
Comet Assay
25%
Programmed Cell Death
25%
phosphorylation
25%
Pharmacology, Toxicology and Pharmaceutical Science
Reactive Oxygen Metabolite
100%
Cigarette Smoke
83%
Premature Labor
50%
Acetic Acid Derivative
33%
Antioxidant
33%
Enzymes
33%
Cysteine
33%
Cell Culture
16%
Base
16%
Phosphotransferase
16%
Western Blot
16%
DNA Base
16%
Fluorescein
16%
Comet Assay
16%
Pregnancy Complication
16%
Etiology
16%
Keyphrases
Fragment Length Analysis
40%