Keyphrases
Genomic Deletion
100%
Ion Channels
100%
Proteomic Changes
100%
Fn14
100%
Sex-specific
100%
Schizophrenia
50%
Aldehyde Dehydrogenase 2 (ALDH2)
50%
Disease Mechanisms
50%
PRKAR2B
50%
Schizophrenia Models
25%
Aetiology of Schizophrenia
25%
Receptor Signaling
25%
Behavioral Phenotype
25%
Protein Kinase
25%
Synaptic Transmission
25%
Proteome
25%
Hippocampus
25%
Alternative Pathway
25%
Neurotransmission
25%
Neuropsychiatric Disorders
25%
Synaptic Dysfunction
25%
Differentially Expressed Proteins
25%
Molecular Characteristics
25%
Underlying Etiologies
25%
Antipsychotics
25%
Dopamine D2 Receptor (DRD2)
25%
γ-aminobutyric Acid
25%
Generating Hypothesis
25%
Same-sex
25%
Endophenotype
25%
Label-free Quantitative Proteomics
25%
Enriched Pathways
25%
Ion Channel Regulation
25%
Genome-wide Association Data
25%
Synaptic Behavior
25%
Biochemistry, Genetics and Molecular Biology
Proteomics
100%
Gene Deletion
100%
Fibroblast Growth Factor
100%
Ion Channel
100%
Synaptic Transmission
66%
ALDH1A1
66%
Proteome
33%
Protein Kinase A
33%
Quantitative Proteomics
33%
Typical Antipsychotic
33%
Endophenotype
33%
Dopamine Receptor D2
33%
Gamma-Aminobutyric Acid
33%
Neuroscience
Ion Channel
100%
Proteomics
100%
Fibroblast Growth Factor 14
100%
Gene Deletion
100%
Synaptic Transmission
66%
Behavior (Neuroscience)
33%
Etiology of Schizophrenia
33%
Gamma-Aminobutyric Acid
33%
Hippocampus
33%
Typical Antipsychotic
33%
Protein Kinase A
33%
Dopamine Receptor D2
33%
Endophenotype
33%