Sex-specific proteomic changes induced by genetic deletion of fibroblast growth factor 14 (FGF14), a regulator of neuronal ion channels

Keyphrases

• Genomic Deletion 100%
• Ion Channels 100%
• Proteomic Changes 100%
• Fn14 100%
• Sex-specific 100%
• Schizophrenia 50%
• Aldehyde Dehydrogenase 2 (ALDH2) 50%
• Disease Mechanisms 50%
• PRKAR2B 50%
• Schizophrenia Models 25%
• Aetiology of Schizophrenia 25%
• Receptor Signaling 25%
• Behavioral Phenotype 25%
• Protein Kinase 25%
• Synaptic Transmission 25%
• Proteome 25%
• Hippocampus 25%
• Alternative Pathway 25%
• Neurotransmission 25%
• Neuropsychiatric Disorders 25%
• Synaptic Dysfunction 25%
• Differentially Expressed Proteins 25%
• Molecular Characteristics 25%
• Underlying Etiologies 25%
• Antipsychotics 25%
• Dopamine D2 Receptor (DRD2) 25%
• γ-aminobutyric Acid 25%
• Generating Hypothesis 25%
• Same-sex 25%
• Endophenotype 25%
• Label-free Quantitative Proteomics 25%
• Enriched Pathways 25%
• Ion Channel Regulation 25%
• Genome-wide Association Data 25%
• Synaptic Behavior 25%

Biochemistry, Genetics and Molecular Biology

• Proteomics 100%
• Gene Deletion 100%
• Fibroblast Growth Factor 100%
• Ion Channel 100%
• Synaptic Transmission 66%
• ALDH1A1 66%
• Proteome 33%
• Protein Kinase A 33%
• Quantitative Proteomics 33%
• Typical Antipsychotic 33%
• Endophenotype 33%
• Dopamine Receptor D2 33%
• Gamma-Aminobutyric Acid 33%

Neuroscience

• Ion Channel 100%
• Proteomics 100%
• Fibroblast Growth Factor 14 100%
• Gene Deletion 100%
• Synaptic Transmission 66%
• Behavior (Neuroscience) 33%
• Etiology of Schizophrenia 33%
• Gamma-Aminobutyric Acid 33%
• Hippocampus 33%
• Typical Antipsychotic 33%
• Protein Kinase A 33%
• Dopamine Receptor D2 33%
• Endophenotype 33%