Tau Loss of Function, by Deletion or Aggregation, Contributes to Peripheral Insulin Resistance

Keyphrases

• Loss Function 100%
• Peripheral Insulin Resistance 100%
• Alzheimer's Disease 80%
• Insulin Signaling 80%
• Primary Hepatocytes 60%
• Insulin Sensitivity 40%
• Type 2 Diabetes Mellitus (T2DM) 40%
• Transgenic Mouse Model 40%
• P38 Mitogen-activated Protein Kinase (p38 MAPK) 40%
• Tau Oligomers 40%
• P301L 40%
• Wild Mice 20%
• Risk Factors 20%
• Signaling Molecules 20%
• Insulin Resistance 20%
• Dose Effect 20%
• Biochemical Analysis 20%
• Epidemiological Data 20%
• Glucose Uptake 20%
• Cellular Level 20%
• Alzheimer's Disease Patient 20%
• C57BL 20%
• Resistance Diseases 20%
• Peripheral Insulin Sensitivity 20%
• Toxic Concentration 20%
• Brain Insulin Resistance 20%
• Alzheimer's Type 20%
• Glucose Uptake Assay 20%

Medicine and Dentistry

• Insulin Resistance 100%
• Tau 100%
• Alzheimer's Disease 55%
• Insulin Signaling 44%
• Insulin Sensitivity 33%
• Hepatocyte 33%
• Transgenic Mouse 22%
• Maturity Onset Diabetes of the Young 22%
• Glucose Uptake 22%
• p38 Mitogen-Activated Protein Kinase 22%
• Oligomer 22%
• Downregulation 11%
• Signaling Protein 11%
• C57BL/6 11%
• Uptake Assay 11%
• Biochemical Analysis 11%

Biochemistry, Genetics and Molecular Biology

• Insulin Resistance 100%
• Tau 100%
• Insulin Signaling 44%
• Hepatocyte 33%
• Insulin Sensitivity 33%
• Mouse Model 22%
• Transgenic Mouse 22%
• Glucose Transport 22%
• P38 Mitogen-Activated Protein Kinases 22%
• Wild Type 11%
• Downregulation 11%
• Wild Type Mouse 11%
• Signaling Protein 11%
• Epidemiological Data 11%
• Toxic Concentration 11%