The L-type calcium channel blocker nimodipine mitigates cytoskeletal proteins phosphorylation in dichlorvos-induced delayed neurotoxicity in rats

Keyphrases

• Cytoskeletal Proteins 100%
• Nimodipine 100%
• Protein Phosphorylation 100%
• Dichlorvos 100%
• L-type Calcium Channel Blocker 100%
• Delayed Neurotoxicity 100%
• Calcium Channel Blockers 33%
• Organophosphate 33%
• Microtubule-associated Protein 33%
• Neurotoxicity 16%
• Attenuation 16%
• Protein Kinase 16%
• Phosphorylation 16%
• Ca2+ 16%
• Western Blotting 16%
• Tubulin 16%
• Protective Efficacy 16%
• Autoradiography 16%
• New Therapeutic Agents 16%
• Hyperphosphorylation 16%
• Calmodulin 16%
• CAMP-dependent Protein Kinase 16%
• Axonal Degeneration 16%
• Neuronal Microtubules 16%
• Electron Microscopic Study 16%
• Exposure Interval 16%
• Microtubule-associated Protein 2 (MAP2) 16%

Neuroscience

• L-Type Calcium Channel 100%
• Protein Phosphorylation 100%
• Nimodipine 100%
• Calcium Channel Blocker 100%
• Delayed Neurotoxicity 100%
• Cytoskeleton Protein 100%
• Protein Kinases 40%
• Organophosphate 40%
• Microtubule Associated Protein 40%
• Neurotoxicity 20%
• Cyclic Adenosine Monophosphate 20%
• Western Blot 20%
• Hyperphosphorylation 20%
• Autoradiograph 20%
• Calmodulin 20%
• Nerve Fiber Degeneration 20%
• Axonal Degeneration 20%
• Microtubule Associated Protein 2 20%
• Tubulin 20%
• Microtubule 20%

Pharmacology, Toxicology and Pharmaceutical Science

• Cytoskeleton Protein 100%
• Nimodipine 100%
• Calcium Channel Blocker 100%
• Delayed Neurotoxicity 100%
• Dichlorvos 100%
• Protein Kinases 33%
• Microtubule Associated Protein 33%
• Organophosphate 33%
• Calcium Ion 16%
• Western Blot 16%
• Neurotoxicity 16%
• Autoradiography 16%
• Calmodulin 16%
• Nerve Fiber Degeneration 16%
• Microtubule Associated Protein 2 16%
• Subcutaneous Injection 16%
• Tubulin 16%