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The pulmonary lesion of smoke inhalation in an ovine model
D. L. Traber
, D. N. Herndon
, M. D. Stein
, J. T. Flynn
, G. D. Niehaus
Research output
:
Contribution to journal
›
Article
›
peer-review
72
Scopus citations
Overview
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Keyphrases
Ovine Model
100%
Smoke Inhalation
100%
Lymph
100%
Pulmonary Lesions
100%
Lung
66%
Sham
66%
Eicosanoids
33%
Major Metabolites
33%
Sheep
33%
Inhalation Injury
33%
Vascular Permeability
33%
Polymorphonuclear Leukocytes
33%
Neutrophils
33%
PaO2
33%
Pulmonary Injury
33%
Cytotoxic Agents
33%
Pulmonary Edema
33%
Prostaglandins F
33%
Prostacyclin
33%
Keto
33%
Temporal Change
33%
Cardiac Index
33%
Pulmonary Artery Pressure
33%
Protein-rich
33%
Tracheobronchial Tree
33%
Insufflated
33%
Plasma Oncotic Pressure
33%
Left Atrial Pressure
33%
Pressure Ratio
33%
Cotton Cloth
33%
Antiprotease Activity
33%
Pharmacology, Toxicology and Pharmaceutical Science
Ovine Model
100%
Proteinase Inhibitor
100%
Lung Edema
100%
Prostaglandin F
100%
Prostacyclin
100%
Icosanoid
100%
Lung Lesion
100%
Cytotoxic Agent
100%