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The role of CXCL10 in the pathogenesis of experimental septic shock
Daniela S. Herzig
, Liming Luan
, Julia K. Bohannon
,
Tracy E. Toliver-Kinsky
, Yin Guo
, Edward R. Sherwood
Anesthesiology
Research output
:
Contribution to journal
›
Article
›
peer-review
40
Scopus citations
Overview
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Keyphrases
Septic Shock
100%
CXCL10
100%
Immunoglobulin G
46%
Cecal Ligation
30%
Cecal Puncture
30%
Knock-in Mouse Model
23%
Leukocyte Trafficking
15%
Inflammation
7%
Gene-deficient Mice
7%
Macrophages
7%
Lung
7%
Interleukin-6
7%
Lymphocyte Activation
7%
High Concentration
7%
Chemokine Receptor CXCR3
7%
Improved Survival
7%
Bacterial Clearance
7%
Acute Phase
7%
Cytokine Concentration
7%
Inhibitory Proteins
7%
Therapeutic Target
7%
Bacterial Burden
7%
Broad-spectrum Antibiotics
7%
Natural Killer Cell Activity
7%
Hypothermia
7%
Lactated Ringer's Solution
7%
Core Body Temperature
7%
Specific Immunoglobulins
7%
Bacterial Survival
7%
Peritoneal Cavity
7%
Leukocyte Recruitment
7%
Plasma Cytokines
7%
Chemokine CXCL10
7%
Pharmacology, Toxicology and Pharmaceutical Science
Immunoglobulin G
100%
Experimental Septic Shock
100%
Septic Shock
66%
Knockout Mouse
50%
Cilastatin Plus Imipenem
33%
Infection
16%
Inflammation
16%
Cytokine
16%
Antibiotic Agent
16%
Interleukin 6
16%
Ringer Lactate Solution
16%
Bacterial Clearance
16%
Gamma Interferon Inducible Protein 10
16%
Hypothermia
16%
Chemokine Receptor CXCR3
16%
Immunology and Microbiology
CXCL10
100%
Immunoglobulin G
46%
Blood Plasma
23%
Knockout Mouse
23%
Cytokine
7%
Macrophage
7%
Leukocyte
7%
Cell Activation
7%
Solution and Solubility
7%
Bacterial Load
7%
Chemokine Receptor CXCR3
7%
Interleukin 6
7%
Natural Killer Cell
7%
Bacterial Clearance
7%
Gamma Interferon Inducible Protein 10
7%
Bacterial Survival
7%
Peritoneal Cavity
7%
Core Temperature
7%
Lymphocyte
7%
Infection
7%